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Anadrol possesses an anabolic rating of 320 (a little more than 3 times the strength of Testosterone, with its anabolic rating of 100)and anandamide concentration of 14.2 ng/ml. The effects on the male reproductive system, however, are only partially known. It is believed that this compound does not possess its own aromatase function, but that it interacts with the aromatase enzyme and the steroid hormone estrogen. Another possible factor in the low efficacy found in women is the fact that estrogen is usually administered in the morning, while androgen is usually given at night. Both have the potential to interfere with the metabolism of both anabolic and estrogenic steroids. Androgens and aryl hydrocarbon steroids do not display any sexual effects after an oral dose of 10 mg, while estrogenic steroids, if used for a prolonged period, induce the production of oestradiol, which acts as a reproductive hormone. It has been suggested that the female sexual organs are particularly susceptible to the steroid hormone. An estrogen-related reaction to these drugs was seen in a study done on women that showed that they could be significantly more sensitive to androgen and estrogen than to androgens. A study done on a number of women and their partners reported that an acute oral administration of estrogen (the equivalent of 3 mg of testosterone) was capable of inducing a 50-80% increase in endogenous androgen release (and the simultaneous release of endogenous androgens) in 30 male and female participants. In addition to the sexual effects, androgens and androgen-dependent aromatase enzymes cause increased synthesis of estradiol. Estrogen is also the major substrate for the enzyme of aromatase (AR) in the prostate and ovum and is the principal substrate for the enzyme of aromatase (ARb) in the adrenal glands. An androgen-induced increase in endogenous androgens levels could contribute to the formation of polyplastic androgen tubular cells in the testes after an exposure. Because anandamide causes a strong androgenic response to exposure, it could contribute to a testicular response. In the rat, exposure to anandamide resulted in an elevated production of androgens, especially testosterone, in the testes and in the adrenal gland. However, exposure to 10 mg or more could result in a decrease in production of testosterone and in a decrease in the level of the androgen, testosterone. Anandamide in humans, in the form of anandamide hydrochloride, results in a slightly higher amount of androgen in the bloodstream. Since the levels are lower, and the rate of production is more, the amount of Related Article: